Susan C. Nagel

Identification and characterization of novel estrogen receptor-beta-sparing antiprogestins.

Short-chain fatty acids enhance nuclear receptor activity through mitogen-activated protein kinase activation and histone deacetylase inhibition.

The importance of appropriate controls, animal feed, and animal models in interpreting results from low-dose studies of bisphenol A.

Implications for human health of the extensive bisphenol A literature showing adverse effects at low doses: a response to attempts to mislead the public.

Large effects from small exposures. III. Endocrine mechanisms mediating effects of bisphenol A at levels of human exposure.

Estrogen modulates expression of putative housekeeping genes in the mouse uterus.

Aberrant gene expression profile in a mouse model of endometriosis mirrors that observed in women.

Neutralizing TIMP1 restores fecundity in a rat model of endometriosis and treating control rats with TIMP1 causes anomalies in ovarian function and embryo development.

Developmental Exposure to Low Dose Xenoestrogens Alters Femur Length and Tensile Strength in Adult Mice.

The estrogenic endocrine disrupting chemical bisphenol A (BPA) and obesity.